Level One Top Secret Document
Unbelievable Results Fighting
Cancer, Leukemia and Diabetes


Dear David,

My Name is G. N. I am a personal friend of Dr. Sam Chachoua. I testified last year In His Trial in LA.

See related article - Ten Million Awarded AIDS and Cancer Researcher

My site is http://www.rainbowminerals.net
I also posted http://www.remissions.org

I have recently come upon some interesting information that Dr. Chachoua and I our Trying to get out to the public in the treatment of just about all forms of leukemia and cancer. below I listed an article that is actually a level one top secrete document that I have posted on my site we are trying to get this information out to the public as fast as possible. The company I work for just came out with a liquid ionic form of vanadium and we are having some unbelievable results fighting Cancer, Leukemia and diabetes

The FDA is now messing with us looking for an excuse to shut us down.

Warmest regards,
G. N


Parker Hughes Institute scientists develop 24 new drugs containing metal Vanadium

Vanadium effective in fighting cancer cells

May 3, 2000, Saint Paul, Minn. ( In a systematic effort to develop effective drugs with a broad spectrum and potent anti-cancer activity, researchers at the St. Paul, Minn.-based Parker Hughes Institute have developed 24 new drugs containing the metal vanadium. The results of this research are published in this month’s issue of Clinical Cancer Research, the official scientific journal of the American Association of Cancer Research.

These novel drugs were effective against 14 of 14 different cancer cell lines as well as cancer cells taken directly from patients. Targeted cancer cells included those of breast cancer, prostate cancer, testicular cancer, ovarian cancer, colon cancer, brain tumors, leukemia and lymphoma. These new anti-cancer drugs work by causing the cancer cells to commit suicide (or apoptosis) within 4 to 24 hours.

These drugs are now being tested in animal safety studies to identify those that have the best therapeutic index.

References Narla RK, Dong Y, D’Cruz OJ, Navara C, Uckun FM. Bis (4,7-dimethyl-1, 10-phenanthroline) sulfatooxovanadium as a novel apoptosis-inducing anticancer agent. Clinical Cancer Research, 6:1546-56, 2000.

Ghosh P, D’Cruz OJ, Narla RK, Uckun FM. Apoptosis-inducing vanadocene compounds against human testicular cancer. Clinical Cancer Research, 6:1536-45, 2000.

The Parker Hughes Institute http://www.parkerhughes.org/, located in Roseville, Minnesota, is a non-profit research organization dedicated to combating cancer, AIDS, and diseases of the immune system.

New agents show potential for treatment of testicular cancer

August 10, 2000, St. Paul, MN...Studies conducted at Parker Hughes Institute involving vanadium-containing agents called "vanadocene" show potential for treating testicular cancer. The results of this work are published in this month's issue of Toxicology and Applied Pharmacology.

This agent was previously found to have powerful sperm immobilizing activity, prompting scientists to conduct further research. The present study establishes that vanadocenes are cytotoxic and induce cell death of testicular cancer cells.

Testicular cancer develops in one or both testicles in men or young boys. The American Cancer Society estimates that in the year 2000 about 6,900 new cases of testicular cancer will be diagnosed in the United States. While testicular cancer is a highly treatable and usually curable form of cancer, an estimated 300 men will die of testicular cancer in this year.

Over 90% of cancers of the testicle develop in certain cells known as germ cells. Most invasive testicular germ cell cancers begin as a noninvasive form of the disease called carcinoma in situ (CIS) or intratubular germ cell neoplasia. Researchers have estimated that it takes approximately five years for CIS to progress to the invasive form of germ cell cancer. When a cancer becomes invasive, its cells have penetrated the surrounding tissues and may have spread through either the blood circulation or the lymph nodes to other parts of the body.

Vanadocenes have the ability to induce selective cytotoxicity and cell death of testicular germ cells. "We are very excited about the potential of vanadocenes to target germ cells," said Dr. Osmond D'Cruz, director, department of reproductive biology, Parker Hughes Institute, and lead author on the study. "This knowledge provides the basis for evaluation of vanadocenes as less-toxic alternatives to currently available treatments for testicular cancer."

Reference: D'Cruz OJ, Uckun FM. Vanadocene-Mediated in Vivo Male Germ Cell Apoptosis. Toxicology and Applied Pharmacology, 166(3):186-95, 2000.

The Parker Hughes Institute http://www.parkerhughes.org/, located in St. Paul, Minnesota, is a non-profit research organization dedicated to eradicating cancer, AIDS, and diseases of the immune system.

Vanadium chemoprevention of 7,12-dimethylbenz(a)anthracene-induced rat mammary carcinogenesis: probable involvement of representative hepatic phase I and II xenobiotic metabolizing enzymes.

Bishayee A, Oinam S, Basu M, Chatterjee M.

Department of Pharmaceutical Technology, Jadavpur University, Calcutta, India. bishayan@umdnj.edu

Vanadium, a non-platinum group metal and dietary micronutrient, is now proving to act as a promising antitumor agent. The present study was conducted to ascertain its antineoplastic potential against an experimental mammary carcinogenesis. Female Sprague-Dawley rats, at 50 days of age, were treated with 7,12-dimethylbenz(a)anthracene (DMBA) (0.5 mg/100 g body weight) by a single tail vein injection in an oil emulsion. Vanadium (ammonium monovanadate) at the concentration of 0.5 ppm was supplemented in drinking water and given ad libitum to the experimental group immediately after the carcinogen treatment and it continued until the termination of the study (24 weeks for histological and biochemical observations and 35 weeks for morphological findings).

It was found that vanadium treatment brought about a substantial protection against DMBA-induced mammary carcinogenesis. This was evident from histological findings that showed no sign of hyperplasia or abnormality after vanadium treatment. There was a significant reduction in incidence (P < 0.05), total number, multiplicity (P < 0.01) and size of palpable mammary tumors and delay in mean latency period of tumor appearance (P < 0.001) following vanadium supplementation compared to DMBA control. From the cumulative results of various hepatic biochemical indices namely, lipid peroxidation, reduced glutathione level, superoxide dismutase activity, cytochrome P450 content and glutathione S-transferase activity, the anticarcinogenic potential of vanadium was well reflected through stabilization of these parameters. Results of the study indicate that the anticarcinogenic activity of vanadium during DMBA-initiated mammary carcinogenesis is mediated through alteration of hepatic antioxidant status as well as modulation of phase I and II drug metabolizing enzymes. On the basis of the observed results, vanadium can be considered as a readily available, promising and novel cancer chemopreventive agent.


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